Data Integration Programmer
|Anna-Kristin’s focus at Blue Brain is domain design, domain documentation and integration of Neuroscience data into the Blue Brain Nexus platform.
Before joining Blue Brain, she was an Electrophysiologist at the MRC Brain Network Dynamics Unit, Oxford. Her role involved investigating the intact and compromised midbrain dopamine system in rodents.
Anna-Kristin has a PhD and Masters in Neuroscience from the University of Oxford, UK and a Bachelors in Biology from the Ludwig Maximilian University of Munich, Germany. She received German Academic Scholarship Foundation and Medical Research Council Scholarships for her doctoral studies and the Max Weber Programme Scholarship for her undergraduate and graduate studies.
In her spare time, she enjoys hiking, reading and listening to music and radio programmes.
Gensberger, K.D., Kaufmann, A.-K., Dietrich, H., Branoner, F., Banchi, R., Chagnaud, B.P., and Straka, H. (2016). Galvanic Vestibular Stimulation: Cellular Substrates and Response Patterns of Neurons in the Vestibulo-Ocular Network. J. Neurosci. 36, 9097–9110. DOI: doi.org/10.1523/JNEUROSCI.4239-15.2016
Marques-Smith, A., Lyngholm, D., Kaufmann, A.-K., Stacey, J.A., Hoerder-Suabedissen, A., Becker, E.B.E., Wilson, M.C., Molnár, Z., and Butt, S.J.B. (2016). A Transient Translaminar GABAergic Interneuron Circuit Connects Thalamocortical Recipient Layers in Neonatal Somatosensory Cortex. Neuron 89, 536–549. DOI: doi.org/10.1016/j.neuron.2016.01.015
Pristerà, A., Lin, W., Kaufmann, A.-K., Brimblecombe, K.R., Threlfell, S., Dodson, P.D., Magill, P.J., Fernandes, C., Cragg, S.J., and Ang, S.-L. (2015). Transcription factors FOXA1 and FOXA2 maintain dopaminergic neuronal properties and control feeding behavior in adult mice. Proc. Natl. Acad. Sci. 112, E4929-38. DOI: doi.org/10.1073/pnas.1503911112
Sloan, M., Alegre-Abarrategui, J., Potgieter, D., Kaufmann, A.-K., Exley, R., Deltheil, T., Threlfell, S., Connor-Robson, N., Brimblecombe, K., Wallings, R., et al. (2016). LRRK2 BAC transgenic rats develop progressive, L-DOPA-responsive motor impairment, and deficits in dopamine circuit function. Hum. Mol. Genet. 25, 951–963. DOI: doi.org/10.1093/hmg/ddv628